Erratum: XI-006 induces potent p53-independent apoptosis in Ewing sarcoma

نویسندگان

  • Kathleen I. Pishas
  • Alaknanda Adwal
  • Susan J. Neuhaus
  • Mark T. Clayer
  • Gelareh Farshid
  • Alexander H. Staudacher
  • David F. Callen
چکیده

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XI-006 induces potent p53-independent apoptosis in Ewing sarcoma

There is an imperious need for the development of novel therapeutics for the treatment of Ewing sarcoma, the second most prevalent solid bone tumour observed in children and young adolescents. Recently, a 4-nitrobenzofuroxan derivative, XI-006 (NSC207895) was shown to diminish MDM4 promoter activity in breast cancer cell lines. As amplification of MDM4 is frequently observed in sarcomas, this s...

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Nutlin-3a is a potential therapeutic for ewing sarcoma.

PURPOSE Although mutations in the TP53 gene occur in half of all cancers, approximately 90% of Ewing sarcomas retain a functional wild-type p53. The low frequency of TP53 alterations in Ewing sarcoma makes this tumor type an ideal candidate for p53-targeted therapies. In this study, we have examined the molecular and cellular responses of cultured Ewing sarcoma cell lines following exposure to ...

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Cancer Therapy: Preclinical Nutlin-3a Is a Potential Therapeutic for Ewing Sarcoma

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Targeting the p53 Pathway in Ewing Sarcoma

The p53 tumour suppressor plays a pivotal role in the prevention of oncogenic transformation. Cancers frequently evade the potent antitumour surveillance mechanisms of p53 through mutation of the TP53 gene, with approximately 50% of all human malignancies expressing dysfunctional, mutated p53 proteins. Interestingly, genetic lesions in the TP53 gene are only observed in 10% of Ewing Sarcomas, w...

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عنوان ژورنال:

دوره 5  شماره 

صفحات  -

تاریخ انتشار 2015